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dc.contributor.authorStembridge, Mike
dc.contributor.authorLevine, Benjamin
dc.date.accessioned2019-03-20T11:52:08Z
dc.date.available2019-03-20T11:52:08Z
dc.date.issued2019-03-24
dc.identifier.citationStembridge, M. and Levine, B.D. (2019) 'No heartbreak at high altitude; preserved cardiac function in chronic hypoxia', Experimental Physiology. https://doi.org/10.1113/EP087665
dc.identifier.issn1469-445X
dc.identifier.urihttp://hdl.handle.net/10369/10387
dc.descriptionArticle published in Experimental Physiology available at https://doi.org/10.1113/EP087665en_US
dc.description.abstractHigh altitude hypoxia presents a series of challenges to the human heart due to concomitant changes in preload, afterload and contractility. This challenge is characterised by a decrease in blood volume due to plasma volume constriction, an increase in right ventricular afterload via hypoxic pulmonary vasoconstriction, and an increase in sympathetic nerve activity . As such, understanding how the heart adapts to this multifaceted challenge has been a topic of interest to physiologists and clinicians for decades. In the current issue of Experimental Physiology, Maufrais et al. (2019) use modern speckle tracking technology to investigate region-specific cardiac performance in chronic hypoxia.en_US
dc.language.isoenen_US
dc.publisherWileyen_US
dc.relation.ispartofseriesExperimental Physiology;
dc.titleNo heartbreak at high altitude; preserved cardiac function in chronic hypoxiaen_US
dc.typeArticleen_US
dc.identifier.doihttps://doi.org/10.1113/EP087665
dcterms.dateAccepted2019-03-08
rioxxterms.funderCardiff Metropolitan Universityen_US
rioxxterms.identifier.projectCardiff Metropolian (Internal)en_US
rioxxterms.versionAMen_US
rioxxterms.licenseref.urihttp://www.rioxx.net/licenses/under-embargo-all-rights-reserveden_US
rioxxterms.licenseref.startdate2019-03-20
rioxxterms.freetoread.startdate2020-03-24
rioxxterms.funder.project37baf166-7129-4cd4-b6a1-507454d1372een_US


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