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dc.contributor.authorMoncada, S.
dc.contributor.authorErusalimsky, Jorge
dc.date.accessioned2008-10-17T11:25:33Z
dc.date.available2008-10-17T11:25:33Z
dc.date.issued2002en_UK
dc.identifier.citationMoncada, S. and Erusalimsky, J.D. (2002) 'OPINION: Does nitric oxide modulate mitochondrial energy generation and apoptosis?', Nature reviews. Molecular cell biology, 3(3), p.214.en_UK
dc.identifier.issn1471-0072en_UK
dc.identifier.urihttp://hdl.handle.net/10369/323
dc.description.abstractThe physiological role of nitric oxide (NO) in the maintenance of vascular tone, in synaptic transmission and in cellular defence is now firmly established. Recent evidence indicates that NO can also affect mitochondrial function. Here, we review findings indicating that NO — through its interaction with components of the electron-transport chain — might function not only as a physiological regulator of cell respiration, but also to augment the generation of reactive oxygen species by mitochondria, and thereby trigger mechanisms of cell survival or death.
dc.publisherNatureen_UK
dc.relation.ispartofseriesNature Reviews Molecular Cell Biologyen_UK
dc.titleOPINION: Does nitric oxide modulate mitochondrial energy generation and apoptosis?en_UK
dc.identifier.doihttp://dx.doi.org/10.1038/nrm762en_UK


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