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dc.contributor.authorShave, Rob
dc.contributor.authorBaggish, Aaron
dc.contributor.authorGeorge, Keith
dc.contributor.authorWood, Malissa
dc.contributor.authorScharhag, Jurgen
dc.contributor.authorWhyte, Greg
dc.contributor.authorGaze, David
dc.contributor.authorThompson, Paul D.
dc.date.accessioned2013-07-03T11:36:31Z
dc.date.available2013-07-03T11:36:31Z
dc.date.issued2010
dc.identifier.citationShave, R., Baggish, A., George, K., Wood, M., Scharhag, J., Whyte, G., Gaze, D. and Thompson, P.D. (2010) 'Exercise-induced cardiac troponin elevation: evidence, mechanisms, and implications', Journal of the American College of Cardiology, 56(3), pp.169-176en_US
dc.identifier.issn0735-1097
dc.identifier.urihttp://hdl.handle.net/10369/4391
dc.description.abstractRegular physical exercise is recommended for the primary prevention of cardiovascular disease. Although the high prevalence of physical inactivity remains a formidable public health issue, participation in exercise programs and recreational sporting events, such as marathons and triathlons, is on the rise. Although regular exercise training reduces cardiovascular disease risk, recent studies have documented elevations in cardiac troponin (cTn) consistent with cardiac damage after bouts of exercise in apparently healthy individuals. At present, the prevalence, mechanism(s), and clinical significance of exercise-induced cTn release remains incompletely understood. This paper will review the biochemistry, prevalence, potential mechanisms, and management of patients with exercise-induced cTn elevations.
dc.language.isoenen_US
dc.relation.ispartofseriesJournal of the American College of Cardiology;
dc.subjectCardiac biomarkersen_US
dc.subjectCardiac damageen_US
dc.subjectExerciseen_US
dc.subjectTroponinen_US
dc.titleExercise-induced cardiac troponin elevation: evidence, mechanisms, and implicationsen_US
dc.typeArticleen_US
dc.identifier.doihttp://dx.doi.org/10.1016/j.jacc.2010.03.037


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