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dc.contributor.authorNightingale, Angus K.
dc.contributor.authorJames, Philip
dc.contributor.authorMorris-Thurgood, Jayne
dc.contributor.authorHarrold, Fraser
dc.contributor.authorTong, Richard
dc.contributor.authorJackson, Simon K.
dc.contributor.authorCockcroft, John R.
dc.date.accessioned2008-10-17T11:31:07Z
dc.date.available2008-10-17T11:31:07Z
dc.date.issued2001-03-01en
dc.identifier.citationAmerican Journal of Physiology - Heart and Circulatory Physiology, 280, pp.H1334-H1339en
dc.identifier.issn03636135
dc.identifier.urihttp://ajpheart.physiology.org/content/280/3/H1334#page
dc.identifier.urihttp://hdl.handle.net/10369/590
dc.description.abstractOne of the key factors leading to cardiovascular disease is endothelial dysfunction, but the mechanisms leading to endothelial dysfunction are complex and poorly understood. This paper provided an insight into the mechanisms of acute endothelial dysfunction following oral methionine loading and has implications in understanding endothelial dysfunction in cardiovascular disease, in particular atherosclerosis. This research, which has implications for cardiovascular health, was supported by the British Heart Foundation and undertaken at the Welsh Heart Research Institute in Cardiff. Tong was involved in the study design, recruitment of subjects and editing the manuscript.en
dc.publisherAmerican Physiological Societyen
dc.relation.ispartofseriesAmerican Journal of Physiology - Heart and Circulatory Physiologyen
dc.titleEvidence against oxidative stress as mechanism of endothelial dysfunction in methionine loading modeen


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